Eidos Therapeutics Inc (NASDAQ:EIDX) insider Jonathan C. Fox sold 5,000 shares of the business’s stock in a transaction that occurred on Monday, April 15th. The stock was sold at an average price of $23.11, for a total value of $115,550.00. The sale was disclosed in a filing with the SEC, which is available through this link.
Shares of EIDX opened at $23.38 on Thursday. The company has a market cap of $919.12 million and a price-to-earnings ratio of -12.57. Eidos Therapeutics Inc has a 52 week low of $8.89 and a 52 week high of $25.55.
Eidos Therapeutics (NASDAQ:EIDX) last issued its quarterly earnings data on Monday, April 15th. The company reported ($0.27) EPS for the quarter, missing analysts’ consensus estimates of ($0.26) by ($0.01). On average, sell-side analysts predict that Eidos Therapeutics Inc will post -1.3 earnings per share for the current fiscal year.
EIDX has been the subject of a number of recent research reports. BTIG Research initiated coverage on Eidos Therapeutics in a report on Wednesday. They set a “buy” rating for the company. Zacks Investment Research raised Eidos Therapeutics from a “sell” rating to a “hold” rating in a report on Monday. JPMorgan Chase & Co. lowered Eidos Therapeutics from an “overweight” rating to a “neutral” rating and set a $29.00 price objective for the company. in a report on Wednesday, January 23rd. Finally, BMO Capital Markets initiated coverage on Eidos Therapeutics in a report on Friday, February 22nd. They set an “outperform” rating and a $28.00 price objective for the company. Three research analysts have rated the stock with a hold rating and six have issued a buy rating to the stock. Eidos Therapeutics has an average rating of “Buy” and a consensus target price of $27.00.
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Eidos Therapeutics Company Profile
Eidos Therapeutics, Inc, a clinical stage biopharmaceutical company, focuses on developing drugs to treat diseases caused by transthyretin (TTR) amyloidosis (ATTR). It is developing AG10, an orally-administered small molecule designed to stabilize tetrameric TTR, thereby halting at its outset the series of molecular events that give rise to ATTR.
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